New Wonder Drug To Treat All Cancers PARP Inhibitors
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|Very soon an universal drug
to treat all type of cancerous tumors may be developed. Scientists have
claimed a breakthrough in turning a breast cancer to a medicine that treat
all types of cancerous tumors. The focus is on cancer drug therapy based
on class of pharmaceuticals called PARP inhibitors. PARP stands for Poly
ADP ribose polymerase is a protein molecule which plays an active role
in various cellular processes, DNA repair and programmed cell death. PARP
inhibitors are particularly effective in treating hereditary breast cancers.
PARP inhibitors have the ability to zero in on rouge cells and tumors and
kill them selectively without harming the healthy cells. This also means
that the side effects that is normally associated with radiotherapy or
chemotherapy is very minimal with this type of pharmaceuticals.
In those prone to or with
hereditary form of breast cancer there is mutation or flaw in the genes
called BRCA1 and BRCA2. BRCA1 is a gene that produces a type of proteins
called breast cancer type 1 susceptibility protein. They are normally expressed
in the cells of breast and some other organs. Whenever there is a damage
to DNA of the cells in these organs BRCA1 immediately repairs this DNA
damage or if it cannot be repaired it kills the cells. Thus cancerous tumors
are killed at the very early stage itself. The function of BRCA2 gene too
is more or less similar though the molecular structure is different. BRCA2
stands for Breast Cancer 2 susceptibility protein. It's main function is
to repair the chromosomal damage and fix the breaks in the DNA structure.
Thus it can be seen that proper functioning of these two genes are very
important to protect us against breast cancer. Whenever there is a flaw
in these two genes which is mainly due to hereditary reasons the person
is at a huge risk of contracting breast cancer.
In breast cancer tumors with
BRCA mutation drugs that inhibit Poly ADP ribose polymerase (PARP) inhibitors
cause double strand break in the cells. Because of BRCA deficiency these
cannot be repaired. This leads to the death of cancerous cells. Normal
cells do not replicate the way cancer cells do so the normal cells are
not affected by PARP inhibitors.
The same principle is applied
by scientists to use PARP inhibitors to treat tumors which do not have
this genetic flaw but by artificially inducing such a genetic flaw. CDK1
or Cyclin dependent kinase 1 is a protein that is very vital in maintaining
normal cell cycle process. It repairs any damage to the DNA of the cells.
By blocking the CDK1 molecule the tumors are deprived of the DNA repair
mechanism. When the subject is given PARP inhibitors the cancerous tumor
was successfully shrunk. Scientists have effectively treated lung cancer
in rats using this method.
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